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DESCRIBE <http://purl.uniprot.org/SHA-384/74BF1BB7E5BED13ECBFEFE46326CE480AD788BA8DD8A80BE6C1CDAE6A0E849F57AC1B71C21AD77F8033B0D88778BE28D>
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http://purl.uniprot.org/SHA-384/74BF1BB7E5BED13ECBFEFE46326CE480AD788BA8DD8A80BE6C1CDAE6A0E849F57AC1B71C21AD77F8033B0D88778BE28D
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/74BF1BB7E5BED13ECBFEFE46326CE480AD788BA8DD8A80BE6C1CDAE6A0E849F57AC1B71C21AD77F8033B0D88778BE28D
http://www.w3.org/2000/01/rdf-schema#comment
"by repressing autocrine IL-2 production Ikaros ensures that naive CD8(+) T cells remain dependent on licensing by APCs and CD4(+) T cells and it may therefore act as a cell-intrinsic safeguard against inappropriate CTL differentiation"
xsd:string
http://purl.uniprot.org/uniprot/#_FDFB44CCF91A08EB6EE6F4433C1F8014F4567902C755986B8B4FFAB9BB6CA558E45C74EF8DD3BB0607B48D6ED1E1DB98
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/74BF1BB7E5BED13ECBFEFE46326CE480AD788BA8DD8A80BE6C1CDAE6A0E849F57AC1B71C21AD77F8033B0D88778BE28D
http://purl.uniprot.org/uniprot/Q5GGW4
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/74BF1BB7E5BED13ECBFEFE46326CE480AD788BA8DD8A80BE6C1CDAE6A0E849F57AC1B71C21AD77F8033B0D88778BE28D
http://purl.uniprot.org/uniprot/#_Q5GGW4-mappedCitation-24778448
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/74BF1BB7E5BED13ECBFEFE46326CE480AD788BA8DD8A80BE6C1CDAE6A0E849F57AC1B71C21AD77F8033B0D88778BE28D