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http://purl.uniprot.org/SHA-384/76230BEB8CDB619201FFE1BE342CF60BC03249348D33EC3053D4AB0E23243B88A604A3DBE4C248037CCDFD3C28BE1CDAhttp://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/76230BEB8CDB619201FFE1BE342CF60BC03249348D33EC3053D4AB0E23243B88A604A3DBE4C248037CCDFD3C28BE1CDAhttp://www.w3.org/2000/01/rdf-schema#comment"Data suggest that NMDAR plays key role in mediating effect of leptin to modulate function of insulin-secreting cells by promoting AMPK-dependent trafficking of KATP and Kv2.1 channels to plasma membrane. (NMDAR = N-methyl-D-aspartate receptor; AMPK = AMP-activated protein kinase; KATP = ATP-sensitive potassium channel; Kv2.1 = delayed-rectifier potassium channel 1)"xsd:string
http://purl.uniprot.org/uniprot/#_52AB04C160C59F93BAC8CD329BA9BD9FC86DA05BD9875E1C0A74FF47A274926D971F11A2513888BD0356C91496F7905Ehttp://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/76230BEB8CDB619201FFE1BE342CF60BC03249348D33EC3053D4AB0E23243B88A604A3DBE4C248037CCDFD3C28BE1CDA
http://purl.uniprot.org/uniprot/Q14721http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/76230BEB8CDB619201FFE1BE342CF60BC03249348D33EC3053D4AB0E23243B88A604A3DBE4C248037CCDFD3C28BE1CDA
http://purl.uniprot.org/uniprot/#_Q14721-mappedCitation-28768770http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/76230BEB8CDB619201FFE1BE342CF60BC03249348D33EC3053D4AB0E23243B88A604A3DBE4C248037CCDFD3C28BE1CDA