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DESCRIBE <http://purl.uniprot.org/SHA-384/76FB21AED124C74B84C7D197F1009F0256F241C1F3CC64E2F999B16749D798CE3CC9D63AA94C384653E0551F91851A17>
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http://purl.uniprot.org/SHA-384/76FB21AED124C74B84C7D197F1009F0256F241C1F3CC64E2F999B16749D798CE3CC9D63AA94C384653E0551F91851A17
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/76FB21AED124C74B84C7D197F1009F0256F241C1F3CC64E2F999B16749D798CE3CC9D63AA94C384653E0551F91851A17
http://www.w3.org/2000/01/rdf-schema#comment
"these data identify KAP as a major regulator of cell division via interactions with numerous target proteins. aberrant splicing or miR-26a decreases KAP expression to promote glioblastoma growth and invasion."
xsd:string
http://purl.uniprot.org/uniprot/#_DDCC7220016ACD16407C0BAAF9F318BF1D3AEF411828DD994DF169DAB8B3426ABF799D8A09ADD7B1DA95A3E1313B1293
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/76FB21AED124C74B84C7D197F1009F0256F241C1F3CC64E2F999B16749D798CE3CC9D63AA94C384653E0551F91851A17
http://purl.uniprot.org/uniprot/Q9C050
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/76FB21AED124C74B84C7D197F1009F0256F241C1F3CC64E2F999B16749D798CE3CC9D63AA94C384653E0551F91851A17
http://purl.uniprot.org/uniprot/#_Q9C050-mappedCitation-24704824
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/76FB21AED124C74B84C7D197F1009F0256F241C1F3CC64E2F999B16749D798CE3CC9D63AA94C384653E0551F91851A17