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DESCRIBE <http://purl.uniprot.org/SHA-384/78E31403C52B21DB0CAC21D3695FD09C5AA017C955FCBC582FECF60E863834F47C72BD5C2308164FBB782914505EFA49>
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http://purl.uniprot.org/SHA-384/78E31403C52B21DB0CAC21D3695FD09C5AA017C955FCBC582FECF60E863834F47C72BD5C2308164FBB782914505EFA49
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/78E31403C52B21DB0CAC21D3695FD09C5AA017C955FCBC582FECF60E863834F47C72BD5C2308164FBB782914505EFA49
http://www.w3.org/2000/01/rdf-schema#comment
"Our studies demonstrate a primary LQT-3 (Na+ channel) defect not influenced by the KCNH2 polymorphism and drug interactions unique to the proband that correlate with improved management of arrhythmias in the patient."
xsd:string
http://purl.uniprot.org/uniprot/#_285657C90AEE95DABEB3A96C3EBF5A55C726D7C0A2B6484FE410B76F11D98DAC5F7F6DEF2C401D9F782EE87AC6734D3A
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/78E31403C52B21DB0CAC21D3695FD09C5AA017C955FCBC582FECF60E863834F47C72BD5C2308164FBB782914505EFA49
http://purl.uniprot.org/uniprot/Q12809
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/78E31403C52B21DB0CAC21D3695FD09C5AA017C955FCBC582FECF60E863834F47C72BD5C2308164FBB782914505EFA49
http://purl.uniprot.org/uniprot/#_Q12809-mappedCitation-23277474
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/78E31403C52B21DB0CAC21D3695FD09C5AA017C955FCBC582FECF60E863834F47C72BD5C2308164FBB782914505EFA49