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DESCRIBE <http://purl.uniprot.org/SHA-384/7AD1D3A7558027E8FA35FF38B7204E3942CFB1DB54C7107724681BDA9F1F96E105F4CA5222C3B13BA04693F2108A510C>
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http://purl.uniprot.org/SHA-384/7AD1D3A7558027E8FA35FF38B7204E3942CFB1DB54C7107724681BDA9F1F96E105F4CA5222C3B13BA04693F2108A510C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/7AD1D3A7558027E8FA35FF38B7204E3942CFB1DB54C7107724681BDA9F1F96E105F4CA5222C3B13BA04693F2108A510C
http://www.w3.org/2000/01/rdf-schema#comment
"These studies reveal mechanisms through which UNC2 gain-of-function mutations disrupt excitation-inhibition balance in the nervous system."
xsd:string
http://purl.uniprot.org/uniprot/#_6B2735520E96E68C640F5D8B9F719108A54AA7F32131B0E6ED57F74D095AFB430FD31A6884C2114180FDFD1796C837B3
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/7AD1D3A7558027E8FA35FF38B7204E3942CFB1DB54C7107724681BDA9F1F96E105F4CA5222C3B13BA04693F2108A510C
http://purl.uniprot.org/uniprot/G5EFB0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/7AD1D3A7558027E8FA35FF38B7204E3942CFB1DB54C7107724681BDA9F1F96E105F4CA5222C3B13BA04693F2108A510C
http://purl.uniprot.org/uniprot/#_G5EFB0-mappedCitation-31364988
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/7AD1D3A7558027E8FA35FF38B7204E3942CFB1DB54C7107724681BDA9F1F96E105F4CA5222C3B13BA04693F2108A510C