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DESCRIBE <http://purl.uniprot.org/SHA-384/7B42FFF362B5C332ACB2EB32AB8FD38F9DF5E78348E73807DC48BFC589E1FEF0284474F9BE521C1F3D8FAB983ABE57DC>
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http://purl.uniprot.org/SHA-384/7B42FFF362B5C332ACB2EB32AB8FD38F9DF5E78348E73807DC48BFC589E1FEF0284474F9BE521C1F3D8FAB983ABE57DC
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/7B42FFF362B5C332ACB2EB32AB8FD38F9DF5E78348E73807DC48BFC589E1FEF0284474F9BE521C1F3D8FAB983ABE57DC
http://www.w3.org/2000/01/rdf-schema#comment
"A modest genetic increase in ACE impairs myocardial tolerance to ischemia. ACE level plays a critical role in cardiac ischemia through both kinin and angiotensin mediated mechanisms."
xsd:string
http://purl.uniprot.org/uniprot/#_56BAE372F85CD0238C635A493021A36556EFDC8A6746CD1796EA9F7B6EAAF5DCB008C9D87C5E4C54776C3EDB11ED5D0F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/7B42FFF362B5C332ACB2EB32AB8FD38F9DF5E78348E73807DC48BFC589E1FEF0284474F9BE521C1F3D8FAB983ABE57DC
http://purl.uniprot.org/uniprot/Q7TSV8
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/7B42FFF362B5C332ACB2EB32AB8FD38F9DF5E78348E73807DC48BFC589E1FEF0284474F9BE521C1F3D8FAB983ABE57DC
http://purl.uniprot.org/uniprot/#_Q7TSV8-mappedCitation-20667972
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/7B42FFF362B5C332ACB2EB32AB8FD38F9DF5E78348E73807DC48BFC589E1FEF0284474F9BE521C1F3D8FAB983ABE57DC