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DESCRIBE <http://purl.uniprot.org/SHA-384/821F3B435F4F10697FDEDC4F44254D70BACEC084C4CAC62F6615CE56E7E46E3E2B4CCB11A9BE9D3B0F46C6FB953976B0>
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http://purl.uniprot.org/SHA-384/821F3B435F4F10697FDEDC4F44254D70BACEC084C4CAC62F6615CE56E7E46E3E2B4CCB11A9BE9D3B0F46C6FB953976B0
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/821F3B435F4F10697FDEDC4F44254D70BACEC084C4CAC62F6615CE56E7E46E3E2B4CCB11A9BE9D3B0F46C6FB953976B0
http://www.w3.org/2000/01/rdf-schema#comment
"activation of GSK-3beta is a key mediator of the initial phase of acetaminophen-induced liver injury through modulating GCL and Mcl-1 degradation as well as JNK activation in liver"
xsd:string
http://purl.uniprot.org/uniprot/#_C6C3101A6C53F22D0617B210A0FCA17C8EDFC36EE1CEED18B6E5FB200DACE97356A12699D658A1A7B32AFA4A3E48C659
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/821F3B435F4F10697FDEDC4F44254D70BACEC084C4CAC62F6615CE56E7E46E3E2B4CCB11A9BE9D3B0F46C6FB953976B0
http://purl.uniprot.org/uniprot/Q3UNA7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/821F3B435F4F10697FDEDC4F44254D70BACEC084C4CAC62F6615CE56E7E46E3E2B4CCB11A9BE9D3B0F46C6FB953976B0
http://purl.uniprot.org/uniprot/#_Q3UNA7-mappedCitation-20061376
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/821F3B435F4F10697FDEDC4F44254D70BACEC084C4CAC62F6615CE56E7E46E3E2B4CCB11A9BE9D3B0F46C6FB953976B0