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http://purl.uniprot.org/SHA-384/82389387E0C325F1BEA77C762A2B5905E10C394FCA5BA6D9DFBA445C39B0E48BE8240DBB2509B1C9EF4599C4635FE020http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/82389387E0C325F1BEA77C762A2B5905E10C394FCA5BA6D9DFBA445C39B0E48BE8240DBB2509B1C9EF4599C4635FE020http://www.w3.org/2000/01/rdf-schema#comment"Data suggest that KCa3.1 is involved in inflammatory changes in brain during diabetic ketoacidosis; here an inhibitor of KCa3.1 (but not an inhibitor of sodium-potassium-chloride symporter) is effective in reducing brain inflammation (in hippocampus cortex and striatum) due to diabetic ketoacidosis."xsd:string
http://purl.uniprot.org/uniprot/#_21D83258F83AD895F225FDD966526D7BCA6EEBFF71A30041DFE53514D45742DAEAD43492DBEB3F0D655F20BD99417503http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/82389387E0C325F1BEA77C762A2B5905E10C394FCA5BA6D9DFBA445C39B0E48BE8240DBB2509B1C9EF4599C4635FE020
http://purl.uniprot.org/uniprot/Q9QYW1http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/82389387E0C325F1BEA77C762A2B5905E10C394FCA5BA6D9DFBA445C39B0E48BE8240DBB2509B1C9EF4599C4635FE020
http://purl.uniprot.org/uniprot/#_Q9QYW1-mappedCitation-27174668http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/82389387E0C325F1BEA77C762A2B5905E10C394FCA5BA6D9DFBA445C39B0E48BE8240DBB2509B1C9EF4599C4635FE020