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DESCRIBE <http://purl.uniprot.org/SHA-384/83F59ECE33C1B8DB8037418953D0B2F428167B0C11F391A7219B14B86EC9D245568A6EAB67C5335C11C9B7525F7095C4>
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http://purl.uniprot.org/SHA-384/83F59ECE33C1B8DB8037418953D0B2F428167B0C11F391A7219B14B86EC9D245568A6EAB67C5335C11C9B7525F7095C4
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/83F59ECE33C1B8DB8037418953D0B2F428167B0C11F391A7219B14B86EC9D245568A6EAB67C5335C11C9B7525F7095C4
http://www.w3.org/2000/01/rdf-schema#comment
"Deletion of adenylyl cyclase type 9 results in a left ventricular diastolic dysfunction which correlates with reduced Hsp20-associated adenylyl cyclase type 9 activity and a significant 64% reduction in baseline Hsp20 phosphorylation."
xsd:string
http://purl.uniprot.org/uniprot/#_28D9B867EB26D463C6B7BAE31645ECC1F0E67D0FCB78F4CE5F16120DB9D12445B473A001A70435D9C3BECB249F2E5ACE
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/83F59ECE33C1B8DB8037418953D0B2F428167B0C11F391A7219B14B86EC9D245568A6EAB67C5335C11C9B7525F7095C4
http://purl.uniprot.org/uniprot/E9Q706
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/83F59ECE33C1B8DB8037418953D0B2F428167B0C11F391A7219B14B86EC9D245568A6EAB67C5335C11C9B7525F7095C4
http://purl.uniprot.org/uniprot/#_E9Q706-mappedCitation-28717248
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/83F59ECE33C1B8DB8037418953D0B2F428167B0C11F391A7219B14B86EC9D245568A6EAB67C5335C11C9B7525F7095C4