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http://purl.uniprot.org/SHA-384/8411A6E9CB4085DEEAA713F1A8C3B4E928561BDF6E10AC3266E936DC02C33E6227902E8D72EF0A8D6EB66F200811DD01http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/8411A6E9CB4085DEEAA713F1A8C3B4E928561BDF6E10AC3266E936DC02C33E6227902E8D72EF0A8D6EB66F200811DD01http://www.w3.org/2000/01/rdf-schema#comment"SMOC2 was highly expressed in both peritoneal and endometrioma lesions. Considering that the genes studied participate either directly or indirectly in cellular processes that can lead to cell migration angiogenesis and inappropriate invasion it is possible that the deregulation of these genes caused the development and maintenance of ectopic tissue."xsd:string
http://purl.uniprot.org/uniprot/#_05362D9568408A94B197927A808047A40EB22F9A01DA0795B6C1AACE52352AE7E5D9B7FEFE44B4D28B608DCA46FBBCADhttp://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/8411A6E9CB4085DEEAA713F1A8C3B4E928561BDF6E10AC3266E936DC02C33E6227902E8D72EF0A8D6EB66F200811DD01
http://purl.uniprot.org/uniprot/Q9H3U7http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/8411A6E9CB4085DEEAA713F1A8C3B4E928561BDF6E10AC3266E936DC02C33E6227902E8D72EF0A8D6EB66F200811DD01
http://purl.uniprot.org/uniprot/#_Q9H3U7-mappedCitation-28678915http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/8411A6E9CB4085DEEAA713F1A8C3B4E928561BDF6E10AC3266E936DC02C33E6227902E8D72EF0A8D6EB66F200811DD01