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DESCRIBE <http://purl.uniprot.org/SHA-384/8431B2D14F72DE68F6EB54D0A73B052349AA16BF62D659A69654657E944E24F69B915221B32E4B6E0D4D2AF6E992876C>
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http://purl.uniprot.org/SHA-384/8431B2D14F72DE68F6EB54D0A73B052349AA16BF62D659A69654657E944E24F69B915221B32E4B6E0D4D2AF6E992876C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/8431B2D14F72DE68F6EB54D0A73B052349AA16BF62D659A69654657E944E24F69B915221B32E4B6E0D4D2AF6E992876C
http://www.w3.org/2000/01/rdf-schema#comment
"point mutation in syntaxin-1A causes abnormal regulation of neuronal plasticity and vesicle recycling and that the affected syntaxin-1A/CaMKII interaction is essential for normal brain and synaptic functions in vivo."
xsd:string
http://purl.uniprot.org/uniprot/#_A26E009FFAE08036013A3F733D6A61DBA3E115C0000F91DD55ABF1B3910EF1B45F66EF4B22023661C0912BC97C0C79F2
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/8431B2D14F72DE68F6EB54D0A73B052349AA16BF62D659A69654657E944E24F69B915221B32E4B6E0D4D2AF6E992876C
http://purl.uniprot.org/uniprot/Q497P1
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8431B2D14F72DE68F6EB54D0A73B052349AA16BF62D659A69654657E944E24F69B915221B32E4B6E0D4D2AF6E992876C
http://purl.uniprot.org/uniprot/#_Q497P1-mappedCitation-24136198
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8431B2D14F72DE68F6EB54D0A73B052349AA16BF62D659A69654657E944E24F69B915221B32E4B6E0D4D2AF6E992876C