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http://purl.uniprot.org/SHA-384/85B54F869F4097C8D45A560E281B8851F34AD1A9DB2BD7597FFD506FD451F64F6FC56834F965E0542D4779B3DFC097A9http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/85B54F869F4097C8D45A560E281B8851F34AD1A9DB2BD7597FFD506FD451F64F6FC56834F965E0542D4779B3DFC097A9http://www.w3.org/2000/01/rdf-schema#comment"Data report that MKL1 was both sufficient and necessary for p65-dependent pro-inflammatory transcriptional program. MKL1 deficiency erased key histone modifications synonymous with transactivation on p65 target promoters. Specifically MKL1 defined histone H3K4 trimethylation landscape for NF-kappaB dependent transcription."xsd:string
http://purl.uniprot.org/uniprot/#_03E1F2C5527A351AC1CC48858C1C3C8F5455148338A983B24C8A5DB7CB54978F87E6EBC73883C0D7F48FAA7459A189B7http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/85B54F869F4097C8D45A560E281B8851F34AD1A9DB2BD7597FFD506FD451F64F6FC56834F965E0542D4779B3DFC097A9
http://purl.uniprot.org/uniprot/B3KRI5http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/85B54F869F4097C8D45A560E281B8851F34AD1A9DB2BD7597FFD506FD451F64F6FC56834F965E0542D4779B3DFC097A9
http://purl.uniprot.org/uniprot/#_B3KRI5-mappedCitation-28298643http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/85B54F869F4097C8D45A560E281B8851F34AD1A9DB2BD7597FFD506FD451F64F6FC56834F965E0542D4779B3DFC097A9