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DESCRIBE <http://purl.uniprot.org/SHA-384/87C6E21FB9D378DC56A21C9B90B6E4B2CD6FC4D0F1E6AB849350D924FD9F3C1792DB3C1B3990B1F5C7AD27766A141F66>
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http://purl.uniprot.org/SHA-384/87C6E21FB9D378DC56A21C9B90B6E4B2CD6FC4D0F1E6AB849350D924FD9F3C1792DB3C1B3990B1F5C7AD27766A141F66
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/87C6E21FB9D378DC56A21C9B90B6E4B2CD6FC4D0F1E6AB849350D924FD9F3C1792DB3C1B3990B1F5C7AD27766A141F66
http://www.w3.org/2000/01/rdf-schema#comment
"FOXM1 overexpression cells showed increased autophagic flux and higher numbers of autophagosomes. Knockdown of ATG7 beclin-1 or cotreatment with chloroquine partly restored sensitivity to docetaxel in the FOXM1-overexpressing cells"
xsd:string
http://purl.uniprot.org/uniprot/#_5C0E6BA17A01383E9A2D34611AEC87C7EB71C4232C78BE195F5F01C64D16701AC81F3359C126BA25DFC99EFBBDF27BA7
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/87C6E21FB9D378DC56A21C9B90B6E4B2CD6FC4D0F1E6AB849350D924FD9F3C1792DB3C1B3990B1F5C7AD27766A141F66
http://purl.uniprot.org/uniprot/A8K591
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/87C6E21FB9D378DC56A21C9B90B6E4B2CD6FC4D0F1E6AB849350D924FD9F3C1792DB3C1B3990B1F5C7AD27766A141F66
http://purl.uniprot.org/uniprot/#_A8K591-mappedCitation-31738958
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/87C6E21FB9D378DC56A21C9B90B6E4B2CD6FC4D0F1E6AB849350D924FD9F3C1792DB3C1B3990B1F5C7AD27766A141F66