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DESCRIBE <http://purl.uniprot.org/SHA-384/88A556D19EA9F14ACB208FA854C2AB54F7A84A983CEFA4A6790622D0C18BD3BC0D1DB836FC13BC74A643426D8C5E2CBB>
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http://purl.uniprot.org/SHA-384/88A556D19EA9F14ACB208FA854C2AB54F7A84A983CEFA4A6790622D0C18BD3BC0D1DB836FC13BC74A643426D8C5E2CBB
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/88A556D19EA9F14ACB208FA854C2AB54F7A84A983CEFA4A6790622D0C18BD3BC0D1DB836FC13BC74A643426D8C5E2CBB
http://www.w3.org/2000/01/rdf-schema#comment
"an Akt-mediated cell survival pathway is compromised by the diminished availability of PIP2 elicited by pathological levels of Gq activity in cardiomyocytes"
xsd:string
http://purl.uniprot.org/uniprot/#_54718C2A597E9EFFD5C5B43BA96ACCD62D21EB9950FAEC82868BBC4481522F168BECC2502CD8B07757363B496086CF39
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/88A556D19EA9F14ACB208FA854C2AB54F7A84A983CEFA4A6790622D0C18BD3BC0D1DB836FC13BC74A643426D8C5E2CBB
http://purl.uniprot.org/uniprot/P21279
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/88A556D19EA9F14ACB208FA854C2AB54F7A84A983CEFA4A6790622D0C18BD3BC0D1DB836FC13BC74A643426D8C5E2CBB
http://purl.uniprot.org/uniprot/#_P21279-mappedCitation-12900409
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/88A556D19EA9F14ACB208FA854C2AB54F7A84A983CEFA4A6790622D0C18BD3BC0D1DB836FC13BC74A643426D8C5E2CBB