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DESCRIBE <http://purl.uniprot.org/SHA-384/8C3A405CE0C1518B71F2E7FC4ED5B4045A2A830254BE2CDF88DB23DB49C2E7CC107CAB0050053C5784A2EB07E1FD6E72>
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http://purl.uniprot.org/SHA-384/8C3A405CE0C1518B71F2E7FC4ED5B4045A2A830254BE2CDF88DB23DB49C2E7CC107CAB0050053C5784A2EB07E1FD6E72
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/8C3A405CE0C1518B71F2E7FC4ED5B4045A2A830254BE2CDF88DB23DB49C2E7CC107CAB0050053C5784A2EB07E1FD6E72
http://www.w3.org/2000/01/rdf-schema#comment
"This study unveils the presence of SR-A1 CD36 and LOX-1 in aortic valves and suggests potential mechanisms by which they may contribute to the pathological angiogenesis inflammation calcification and lipid accumulation in AVS."
xsd:string
http://purl.uniprot.org/uniprot/#_B07FA951E07922735BF0D5E4934987E113F347F5426EDA20EAA829D60A0FC286AFC3DBB6CCA38B0E22F037DE9E7D1E83
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/8C3A405CE0C1518B71F2E7FC4ED5B4045A2A830254BE2CDF88DB23DB49C2E7CC107CAB0050053C5784A2EB07E1FD6E72
http://purl.uniprot.org/uniprot/Q9H7N4
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8C3A405CE0C1518B71F2E7FC4ED5B4045A2A830254BE2CDF88DB23DB49C2E7CC107CAB0050053C5784A2EB07E1FD6E72
http://purl.uniprot.org/uniprot/#_Q9H7N4-mappedCitation-24929820
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8C3A405CE0C1518B71F2E7FC4ED5B4045A2A830254BE2CDF88DB23DB49C2E7CC107CAB0050053C5784A2EB07E1FD6E72