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DESCRIBE <http://purl.uniprot.org/SHA-384/8D8C879AEA9E00656C229244E01BF60EBECB88CFCC8B4AF9886F2FBD4B6176B9EABA0E227DA8981C0AFAC074AF0888E0>
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http://purl.uniprot.org/SHA-384/8D8C879AEA9E00656C229244E01BF60EBECB88CFCC8B4AF9886F2FBD4B6176B9EABA0E227DA8981C0AFAC074AF0888E0
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/8D8C879AEA9E00656C229244E01BF60EBECB88CFCC8B4AF9886F2FBD4B6176B9EABA0E227DA8981C0AFAC074AF0888E0
http://www.w3.org/2000/01/rdf-schema#comment
"These findings support studies indicating that hypomorphic mutations of human DNAAF2 can result in ciliary dyskinesia and identify Dnaaf2 as an essential component of cilia function in vivo."
xsd:string
http://purl.uniprot.org/uniprot/#_B78BC97883EDAE52E45B79AF367745A3F0ED3D8641D43CAE4D16BA46B3C845246BBAE610D0E48CB51EBBBDEF148D8F09
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/8D8C879AEA9E00656C229244E01BF60EBECB88CFCC8B4AF9886F2FBD4B6176B9EABA0E227DA8981C0AFAC074AF0888E0
http://purl.uniprot.org/uniprot/Q8BPI1
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8D8C879AEA9E00656C229244E01BF60EBECB88CFCC8B4AF9886F2FBD4B6176B9EABA0E227DA8981C0AFAC074AF0888E0
http://purl.uniprot.org/uniprot/#_Q8BPI1-mappedCitation-31107948
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8D8C879AEA9E00656C229244E01BF60EBECB88CFCC8B4AF9886F2FBD4B6176B9EABA0E227DA8981C0AFAC074AF0888E0