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DESCRIBE <http://purl.uniprot.org/SHA-384/8FEDE5C5DBC4B413206FAD704B4D27763134CE7527E989197D3F36615C46612EB0D71617168150FB4AA0C532D75DA3AA>
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http://purl.uniprot.org/SHA-384/8FEDE5C5DBC4B413206FAD704B4D27763134CE7527E989197D3F36615C46612EB0D71617168150FB4AA0C532D75DA3AA
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/8FEDE5C5DBC4B413206FAD704B4D27763134CE7527E989197D3F36615C46612EB0D71617168150FB4AA0C532D75DA3AA
http://www.w3.org/2000/01/rdf-schema#comment
"data supports a mechanism whereby telomerase deficiency and subsequent shortened telomeres initiate a DNA damage response and create a pro-oxidant environment especially in Dyskeratosis Congenita cells carrying TINF2 mutations"
xsd:string
http://purl.uniprot.org/uniprot/#_E49732AAF5D942FE87E4CB466F10F4AB1EBA86CC94A70B54308C6A21C0995E0BD41F6D6E563895FAB2490B94A866520A
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/8FEDE5C5DBC4B413206FAD704B4D27763134CE7527E989197D3F36615C46612EB0D71617168150FB4AA0C532D75DA3AA
http://purl.uniprot.org/uniprot/H0YL20
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8FEDE5C5DBC4B413206FAD704B4D27763134CE7527E989197D3F36615C46612EB0D71617168150FB4AA0C532D75DA3AA
http://purl.uniprot.org/uniprot/#_H0YL20-mappedCitation-26859482
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8FEDE5C5DBC4B413206FAD704B4D27763134CE7527E989197D3F36615C46612EB0D71617168150FB4AA0C532D75DA3AA