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DESCRIBE <http://purl.uniprot.org/SHA-384/8FF5469ED535CF108CFC56B3963E933CB5D688580FCC5DB65C403FC665EAECF16662D90B0CF5AC47BAC33029CB0F7EB4>
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http://purl.uniprot.org/SHA-384/8FF5469ED535CF108CFC56B3963E933CB5D688580FCC5DB65C403FC665EAECF16662D90B0CF5AC47BAC33029CB0F7EB4
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/8FF5469ED535CF108CFC56B3963E933CB5D688580FCC5DB65C403FC665EAECF16662D90B0CF5AC47BAC33029CB0F7EB4
http://www.w3.org/2000/01/rdf-schema#comment
"diapocynin inhibited ROS production abolished iPLA2 activity and reduced Ca2+ influx through stretch-activated and store-operated channels two major pathways responsible for excessive Ca2+ entry in dystrophic muscle"
xsd:string
http://purl.uniprot.org/uniprot/#_117A0A4A8DBDD412C878AB1DF8779F484AB095109833DF4E2677B1C7ACD147D01C87B0CA375795D94DB67BF243CE4B86
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/8FF5469ED535CF108CFC56B3963E933CB5D688580FCC5DB65C403FC665EAECF16662D90B0CF5AC47BAC33029CB0F7EB4
http://purl.uniprot.org/uniprot/P97819
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8FF5469ED535CF108CFC56B3963E933CB5D688580FCC5DB65C403FC665EAECF16662D90B0CF5AC47BAC33029CB0F7EB4
http://purl.uniprot.org/uniprot/#_P97819-mappedCitation-25329652
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/8FF5469ED535CF108CFC56B3963E933CB5D688580FCC5DB65C403FC665EAECF16662D90B0CF5AC47BAC33029CB0F7EB4