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DESCRIBE <http://purl.uniprot.org/SHA-384/918D037D72B0CCCBBD0D16F0001CEB0F17C5E69B13E8B8BC4FA0839BAE8174047EF984835DD93B1D2B04C578EE66587F>
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http://purl.uniprot.org/SHA-384/918D037D72B0CCCBBD0D16F0001CEB0F17C5E69B13E8B8BC4FA0839BAE8174047EF984835DD93B1D2B04C578EE66587F
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/918D037D72B0CCCBBD0D16F0001CEB0F17C5E69B13E8B8BC4FA0839BAE8174047EF984835DD93B1D2B04C578EE66587F
http://www.w3.org/2000/01/rdf-schema#comment
"These results suggest that TCF7L2 variants may act through insulin secretion to increase the risk of type 2 diabetes; Observational study of gene-disease association. (HuGE Navigator)"
xsd:string
http://purl.uniprot.org/uniprot/#_69ABD3BBB6F4BE56462BAAC48F7E3CC80DAFA685A92DAFE2F40CF5934457369AFFED484D677A3BB8E3F8977CA48D237C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/918D037D72B0CCCBBD0D16F0001CEB0F17C5E69B13E8B8BC4FA0839BAE8174047EF984835DD93B1D2B04C578EE66587F
http://purl.uniprot.org/uniprot/Q5VVR7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/918D037D72B0CCCBBD0D16F0001CEB0F17C5E69B13E8B8BC4FA0839BAE8174047EF984835DD93B1D2B04C578EE66587F
http://purl.uniprot.org/uniprot/#_Q5VVR7-mappedCitation-17003358
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/918D037D72B0CCCBBD0D16F0001CEB0F17C5E69B13E8B8BC4FA0839BAE8174047EF984835DD93B1D2B04C578EE66587F