"Exogenous expression of miR-142-5p inhibitor resulted in a significant reduction of viral titer indicating proviral role of miR-142-5p. Functional studies of hsa-miR-142-5p identified its role in transforming growth factor beta (TGFbeta) signalling as TGFbeta receptor 2 and SMAD3 were degraded during both hsa-miR-142-5p overexpression and rotavirus infection."xsd:string