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DESCRIBE <http://purl.uniprot.org/SHA-384/944656DE19D3A6E63DC0C93648E22D5E5C50C02ED207EE44C8E9C9481D8F527691878E5EE77BE68248F158748852638C>
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http://purl.uniprot.org/SHA-384/944656DE19D3A6E63DC0C93648E22D5E5C50C02ED207EE44C8E9C9481D8F527691878E5EE77BE68248F158748852638C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/944656DE19D3A6E63DC0C93648E22D5E5C50C02ED207EE44C8E9C9481D8F527691878E5EE77BE68248F158748852638C
http://www.w3.org/2000/01/rdf-schema#comment
"Results show that amplified signaling of interleukin-17 receptor B (IL-17RB) and its ligand IL-17B promot tumorigenicity in breast cancer cells and impeded acinus formation in immortalized normal mammary epithelial cells."
xsd:string
http://purl.uniprot.org/uniprot/#_5DFAB4C10FF08636FE8CAF566DD9D772E2C6D1DA8354CB474613ABADD096629E8B59BEFDFB33356445824421051F157A
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/944656DE19D3A6E63DC0C93648E22D5E5C50C02ED207EE44C8E9C9481D8F527691878E5EE77BE68248F158748852638C
http://purl.uniprot.org/uniprot/Q6IAG3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/944656DE19D3A6E63DC0C93648E22D5E5C50C02ED207EE44C8E9C9481D8F527691878E5EE77BE68248F158748852638C
http://purl.uniprot.org/uniprot/#_Q6IAG3-mappedCitation-23851503
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/944656DE19D3A6E63DC0C93648E22D5E5C50C02ED207EE44C8E9C9481D8F527691878E5EE77BE68248F158748852638C