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DESCRIBE <http://purl.uniprot.org/SHA-384/95D4E7F7DBD9E8E1021CC7AFFAA470CF5265FAC27A785DAAA4F90952960BEDF69D67778C8D012D216DD59751853A8827>
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http://purl.uniprot.org/SHA-384/95D4E7F7DBD9E8E1021CC7AFFAA470CF5265FAC27A785DAAA4F90952960BEDF69D67778C8D012D216DD59751853A8827
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/95D4E7F7DBD9E8E1021CC7AFFAA470CF5265FAC27A785DAAA4F90952960BEDF69D67778C8D012D216DD59751853A8827
http://www.w3.org/2000/01/rdf-schema#comment
"our data showed that bile acid-activated FXR stimulates miR-22-silenced CCNA2 a novel pathway for FXR to exert its protective effect in the gastrointestinal tract."
xsd:string
http://purl.uniprot.org/uniprot/#_971A5EB0B6FD025170A25C633CF3E7C9F354FA08D537E4D0FF36FBF3C5145432E2223C133FD3487E16B098285FC32BBE
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/95D4E7F7DBD9E8E1021CC7AFFAA470CF5265FAC27A785DAAA4F90952960BEDF69D67778C8D012D216DD59751853A8827
http://purl.uniprot.org/uniprot/F1DAL1
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/95D4E7F7DBD9E8E1021CC7AFFAA470CF5265FAC27A785DAAA4F90952960BEDF69D67778C8D012D216DD59751853A8827
http://purl.uniprot.org/uniprot/#_F1DAL1-mappedCitation-25596928
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/95D4E7F7DBD9E8E1021CC7AFFAA470CF5265FAC27A785DAAA4F90952960BEDF69D67778C8D012D216DD59751853A8827