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DESCRIBE <http://purl.uniprot.org/SHA-384/970009068A8173D1D4922A98DCCA9B096C5EDFA1C4AB7587FFB58146F702CE30F2BEFDAD0FE45E579B1F7B87DAAEA13E>
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http://purl.uniprot.org/SHA-384/970009068A8173D1D4922A98DCCA9B096C5EDFA1C4AB7587FFB58146F702CE30F2BEFDAD0FE45E579B1F7B87DAAEA13E
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/970009068A8173D1D4922A98DCCA9B096C5EDFA1C4AB7587FFB58146F702CE30F2BEFDAD0FE45E579B1F7B87DAAEA13E
http://www.w3.org/2000/01/rdf-schema#comment
"Exploration of the molecular causes of enhanced cardiac hypertrophy revealed significant activation of beta-catenin/GSK-3beta signaling whereas MAPK and MKL1/serum-response factor pathways were inhibited."
xsd:string
http://purl.uniprot.org/uniprot/#_E0F6E1762D6EC6D161B874CEA905EE22F2C2F1AE0EDD9E055C5F356436B61C13F8C66C27527A9E5D8106230223F9EA8F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/970009068A8173D1D4922A98DCCA9B096C5EDFA1C4AB7587FFB58146F702CE30F2BEFDAD0FE45E579B1F7B87DAAEA13E
http://purl.uniprot.org/uniprot/T2HRH7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/970009068A8173D1D4922A98DCCA9B096C5EDFA1C4AB7587FFB58146F702CE30F2BEFDAD0FE45E579B1F7B87DAAEA13E
http://purl.uniprot.org/uniprot/#_T2HRH7-mappedCitation-26719331
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/970009068A8173D1D4922A98DCCA9B096C5EDFA1C4AB7587FFB58146F702CE30F2BEFDAD0FE45E579B1F7B87DAAEA13E