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DESCRIBE <http://purl.uniprot.org/SHA-384/97AD1A0EAF6E6E3EFE5B7BCBDB0F0414EE876EB093C7D8FD55F5268D1CD124A67354F976FB2D9BCDCD08B1D6718C9E3E>
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http://purl.uniprot.org/SHA-384/97AD1A0EAF6E6E3EFE5B7BCBDB0F0414EE876EB093C7D8FD55F5268D1CD124A67354F976FB2D9BCDCD08B1D6718C9E3E
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/97AD1A0EAF6E6E3EFE5B7BCBDB0F0414EE876EB093C7D8FD55F5268D1CD124A67354F976FB2D9BCDCD08B1D6718C9E3E
http://www.w3.org/2000/01/rdf-schema#comment
"These results suggest that modulation of the ST2L internalization by FAK/GSK3beta might serve as a unique strategy to lessen pulmonary inflammation."
xsd:string
http://purl.uniprot.org/uniprot/#_1A88BF4EF93327968101E9784C1C9C2E22DBEE0871FACAE7804A362EBEDAD65B8816DD9187C84AEF7CFAE1074AB2365C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/97AD1A0EAF6E6E3EFE5B7BCBDB0F0414EE876EB093C7D8FD55F5268D1CD124A67354F976FB2D9BCDCD08B1D6718C9E3E
http://purl.uniprot.org/uniprot/P14719
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/97AD1A0EAF6E6E3EFE5B7BCBDB0F0414EE876EB093C7D8FD55F5268D1CD124A67354F976FB2D9BCDCD08B1D6718C9E3E
http://purl.uniprot.org/uniprot/#_P14719-mappedCitation-25472995
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/97AD1A0EAF6E6E3EFE5B7BCBDB0F0414EE876EB093C7D8FD55F5268D1CD124A67354F976FB2D9BCDCD08B1D6718C9E3E