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DESCRIBE <http://purl.uniprot.org/SHA-384/986EEA4395C8390D15F6136BE779AB645AEA44F4E324EB5CE2466EC271E5E680E3F0F29F4800E4ED909CC2816F31823B>
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http://purl.uniprot.org/SHA-384/986EEA4395C8390D15F6136BE779AB645AEA44F4E324EB5CE2466EC271E5E680E3F0F29F4800E4ED909CC2816F31823B
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/986EEA4395C8390D15F6136BE779AB645AEA44F4E324EB5CE2466EC271E5E680E3F0F29F4800E4ED909CC2816F31823B
http://www.w3.org/2000/01/rdf-schema#comment
"The C-type lectin receptor Clec1A plays an important role in the development of experimental autoimmune encephalomyelitis by enhancing antigen presenting ability of dendritic cells and inducing inflammatory cytokine IL-17."
xsd:string
http://purl.uniprot.org/uniprot/#_946B62474EE306E0224F71EDFAAF004D77E9C9CB11305B722E2B954C58BD2B2194AB56F7C8FC974D65F644C3A58D97EB
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/986EEA4395C8390D15F6136BE779AB645AEA44F4E324EB5CE2466EC271E5E680E3F0F29F4800E4ED909CC2816F31823B
http://purl.uniprot.org/uniprot/A0A0N4SV03
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/986EEA4395C8390D15F6136BE779AB645AEA44F4E324EB5CE2466EC271E5E680E3F0F29F4800E4ED909CC2816F31823B
http://purl.uniprot.org/uniprot/#_A0A0N4SV03-mappedCitation-35135958
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/986EEA4395C8390D15F6136BE779AB645AEA44F4E324EB5CE2466EC271E5E680E3F0F29F4800E4ED909CC2816F31823B