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DESCRIBE <http://purl.uniprot.org/SHA-384/9A2908C1580F1085591F70DBED04E3A1871A7A3B8EDFA686406A60E8E3980ADDD4BF84CB525FF3CAAA57617D30C9356A>
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http://purl.uniprot.org/SHA-384/9A2908C1580F1085591F70DBED04E3A1871A7A3B8EDFA686406A60E8E3980ADDD4BF84CB525FF3CAAA57617D30C9356A
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/9A2908C1580F1085591F70DBED04E3A1871A7A3B8EDFA686406A60E8E3980ADDD4BF84CB525FF3CAAA57617D30C9356A
http://www.w3.org/2000/01/rdf-schema#comment
"Results suggest that Nox4 in cardiac myocytes is a major source of mitochondrial oxidative stress thereby mediating mitochondrial and cardiac dysfunction during PO."
xsd:string
http://purl.uniprot.org/uniprot/#_740F1BA7A55E098AC95CE06E276600F2D66133AB781C3C25C8917460942931AC34E0DBE4718BBBBAA48D31C13D767D9D
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/9A2908C1580F1085591F70DBED04E3A1871A7A3B8EDFA686406A60E8E3980ADDD4BF84CB525FF3CAAA57617D30C9356A
http://purl.uniprot.org/uniprot/Q9JHI8
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9A2908C1580F1085591F70DBED04E3A1871A7A3B8EDFA686406A60E8E3980ADDD4BF84CB525FF3CAAA57617D30C9356A
http://purl.uniprot.org/uniprot/#_Q9JHI8-mappedCitation-20713697
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9A2908C1580F1085591F70DBED04E3A1871A7A3B8EDFA686406A60E8E3980ADDD4BF84CB525FF3CAAA57617D30C9356A