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DESCRIBE <http://purl.uniprot.org/SHA-384/9ACE6B95DE8B026BCD48079AA374C9EB41BA4DE4E1B6FBC39B0AC8C103FE81C1E9E0ECD82F3B5E71F2D7752F8ED8AA12>
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http://purl.uniprot.org/SHA-384/9ACE6B95DE8B026BCD48079AA374C9EB41BA4DE4E1B6FBC39B0AC8C103FE81C1E9E0ECD82F3B5E71F2D7752F8ED8AA12
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/9ACE6B95DE8B026BCD48079AA374C9EB41BA4DE4E1B6FBC39B0AC8C103FE81C1E9E0ECD82F3B5E71F2D7752F8ED8AA12
http://www.w3.org/2000/01/rdf-schema#comment
"In this study they investigated whether endogenous panx-1 was activated and contributed to membrane current in osmotically swollen HEK-293 cells."
xsd:string
http://purl.uniprot.org/uniprot/#_D2CD1FCF17A1EC452CD7AD3F1FB1718CDFF1724BC89D099918D85E8ACF2A01B89BBB9B2BAFE025584A3CF243EDC6062B
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/9ACE6B95DE8B026BCD48079AA374C9EB41BA4DE4E1B6FBC39B0AC8C103FE81C1E9E0ECD82F3B5E71F2D7752F8ED8AA12
http://purl.uniprot.org/uniprot/Q96RD7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9ACE6B95DE8B026BCD48079AA374C9EB41BA4DE4E1B6FBC39B0AC8C103FE81C1E9E0ECD82F3B5E71F2D7752F8ED8AA12
http://purl.uniprot.org/uniprot/#_Q96RD7-mappedCitation-19150332
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9ACE6B95DE8B026BCD48079AA374C9EB41BA4DE4E1B6FBC39B0AC8C103FE81C1E9E0ECD82F3B5E71F2D7752F8ED8AA12