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http://purl.uniprot.org/SHA-384/9AF530128239FBA5EE9A816627322A8D5F517C6E6F668CE4098EE046E1B4FB7ECE9A4437129B5B2F23936F1FB4B868A2http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/9AF530128239FBA5EE9A816627322A8D5F517C6E6F668CE4098EE046E1B4FB7ECE9A4437129B5B2F23936F1FB4B868A2http://www.w3.org/2000/01/rdf-schema#comment"Mutations in TP53 and STK11 also impacted tumor biology regardless of KRAS status with TP53 strongly associated with enhanced proliferation and STK11 with suppression of immune surveillance. These findings illustrate the remarkably distinct ways through which tumor suppressor mutations may contribute to heterogeneity in KRAS-mutant tumor biology."xsd:string
http://purl.uniprot.org/uniprot/#_FC80626BC6895C64EB3F01ABA0204109844E4B5B1117D00D0E40A876A15819F14A9BEBF246D3571C40431E408F8ECF5Bhttp://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/9AF530128239FBA5EE9A816627322A8D5F517C6E6F668CE4098EE046E1B4FB7ECE9A4437129B5B2F23936F1FB4B868A2
http://purl.uniprot.org/uniprot/A6YR18http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/9AF530128239FBA5EE9A816627322A8D5F517C6E6F668CE4098EE046E1B4FB7ECE9A4437129B5B2F23936F1FB4B868A2
http://purl.uniprot.org/uniprot/#_A6YR18-mappedCitation-26477306http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/9AF530128239FBA5EE9A816627322A8D5F517C6E6F668CE4098EE046E1B4FB7ECE9A4437129B5B2F23936F1FB4B868A2