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DESCRIBE <http://purl.uniprot.org/SHA-384/9B5A3D06E9E7601CCD20A4CE7FC2ECC9A7CD7AE1FDBCE84F9425792DE3CD3D604AD973E6BB24B8ED857DECA9D9A17E9F>
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http://purl.uniprot.org/SHA-384/9B5A3D06E9E7601CCD20A4CE7FC2ECC9A7CD7AE1FDBCE84F9425792DE3CD3D604AD973E6BB24B8ED857DECA9D9A17E9F
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/9B5A3D06E9E7601CCD20A4CE7FC2ECC9A7CD7AE1FDBCE84F9425792DE3CD3D604AD973E6BB24B8ED857DECA9D9A17E9F
http://www.w3.org/2000/01/rdf-schema#comment
"The Brugada syndrome mutation A39V when introduced into rat brain Cav1.2 does not trigger the loss-of-trafficking phenotype seen in a previous study on the human heart isoform of the channel"
xsd:string
http://purl.uniprot.org/uniprot/#_3ECCB1C0DD4EA40E74DAD75E5C5779EAD5ACD87F471DC783044C031F73323D3948AFE262F364EBB23834C573373CA48E
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/9B5A3D06E9E7601CCD20A4CE7FC2ECC9A7CD7AE1FDBCE84F9425792DE3CD3D604AD973E6BB24B8ED857DECA9D9A17E9F
http://purl.uniprot.org/uniprot/Q71QJ6
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9B5A3D06E9E7601CCD20A4CE7FC2ECC9A7CD7AE1FDBCE84F9425792DE3CD3D604AD973E6BB24B8ED857DECA9D9A17E9F
http://purl.uniprot.org/uniprot/#_Q71QJ6-mappedCitation-22385640
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9B5A3D06E9E7601CCD20A4CE7FC2ECC9A7CD7AE1FDBCE84F9425792DE3CD3D604AD973E6BB24B8ED857DECA9D9A17E9F