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DESCRIBE <http://purl.uniprot.org/SHA-384/9D52318FA0D3E3366EBBF44F045DE40A31E2FCD4A4263B0CEBF250F0663CBDF71714F23239EF962EB2D2A9A19BE45718>
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http://purl.uniprot.org/SHA-384/9D52318FA0D3E3366EBBF44F045DE40A31E2FCD4A4263B0CEBF250F0663CBDF71714F23239EF962EB2D2A9A19BE45718
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/9D52318FA0D3E3366EBBF44F045DE40A31E2FCD4A4263B0CEBF250F0663CBDF71714F23239EF962EB2D2A9A19BE45718
http://www.w3.org/2000/01/rdf-schema#comment
"The findings suggest that the signaling functions of Dystrophin protein are able to ameliorate dilated cardiomyopathy and thus might help to improve heart muscle function in micro-Dystrophin-based gene therapy approaches."
xsd:string
http://purl.uniprot.org/uniprot/#_763D20A3A2113B0B36019371CE8938E76382A29062E1956CC8AD35A88985C6422DFB84328F9DD7A072B19D14F3E774E6
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/9D52318FA0D3E3366EBBF44F045DE40A31E2FCD4A4263B0CEBF250F0663CBDF71714F23239EF962EB2D2A9A19BE45718
http://purl.uniprot.org/uniprot/Q7YU29
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9D52318FA0D3E3366EBBF44F045DE40A31E2FCD4A4263B0CEBF250F0663CBDF71714F23239EF962EB2D2A9A19BE45718
http://purl.uniprot.org/uniprot/#_Q7YU29-mappedCitation-24231130
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9D52318FA0D3E3366EBBF44F045DE40A31E2FCD4A4263B0CEBF250F0663CBDF71714F23239EF962EB2D2A9A19BE45718