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DESCRIBE <http://purl.uniprot.org/SHA-384/9E5DA1A06431E8BF36B8FAB77E157A9C49C13928980CE2E78B27270362BDA87C7D3480B84809E0A2EF5A8B8666BD4B93>
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http://purl.uniprot.org/SHA-384/9E5DA1A06431E8BF36B8FAB77E157A9C49C13928980CE2E78B27270362BDA87C7D3480B84809E0A2EF5A8B8666BD4B93
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/9E5DA1A06431E8BF36B8FAB77E157A9C49C13928980CE2E78B27270362BDA87C7D3480B84809E0A2EF5A8B8666BD4B93
http://www.w3.org/2000/01/rdf-schema#comment
"Results indicate that for human P/Q-type Ca(2+) channels with the long-alpha(1A)-subunit isoform both missense and nonsense episodic ataxia type 2 mutants indeed display prominent dominant-negative effects."
xsd:string
http://purl.uniprot.org/uniprot/#_9ADBB8316775B8BA188C571DD40BCBEFEC46ADD03A425494B776C1826E0D859947075F0E64A0A8CAF776FC321C47572F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/9E5DA1A06431E8BF36B8FAB77E157A9C49C13928980CE2E78B27270362BDA87C7D3480B84809E0A2EF5A8B8666BD4B93
http://purl.uniprot.org/uniprot/Q9UN69
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9E5DA1A06431E8BF36B8FAB77E157A9C49C13928980CE2E78B27270362BDA87C7D3480B84809E0A2EF5A8B8666BD4B93
http://purl.uniprot.org/uniprot/#_Q9UN69-mappedCitation-16306128
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/9E5DA1A06431E8BF36B8FAB77E157A9C49C13928980CE2E78B27270362BDA87C7D3480B84809E0A2EF5A8B8666BD4B93