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DESCRIBE <http://purl.uniprot.org/SHA-384/A1EA6031440A741DEEEC79D82458E441B0812B864695F32EF423842AC711A95AB4856A50D9E197DFEDE836C922859D4B>
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http://purl.uniprot.org/SHA-384/A1EA6031440A741DEEEC79D82458E441B0812B864695F32EF423842AC711A95AB4856A50D9E197DFEDE836C922859D4B
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A1EA6031440A741DEEEC79D82458E441B0812B864695F32EF423842AC711A95AB4856A50D9E197DFEDE836C922859D4B
http://www.w3.org/2000/01/rdf-schema#comment
"Taken together our data propose a novel oncogene-tumor suppressor interplay where oncogenic PFTK1 confers HCC cell motility through inactivating the actin-binding motile suppressing function of TAGLN2 via phosphorylation."
xsd:string
http://purl.uniprot.org/uniprot/#_A7BC62BB81C01FE5A5F84B67CE045F74D6AA9625FFE3C957794674CAE0FDDB2ADFA40784BCC0A28DA582B2E40C0C8A40
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A1EA6031440A741DEEEC79D82458E441B0812B864695F32EF423842AC711A95AB4856A50D9E197DFEDE836C922859D4B
http://purl.uniprot.org/uniprot/O94921
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A1EA6031440A741DEEEC79D82458E441B0812B864695F32EF423842AC711A95AB4856A50D9E197DFEDE836C922859D4B
http://purl.uniprot.org/uniprot/#_O94921-mappedCitation-21577206
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A1EA6031440A741DEEEC79D82458E441B0812B864695F32EF423842AC711A95AB4856A50D9E197DFEDE836C922859D4B