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DESCRIBE <http://purl.uniprot.org/SHA-384/A226B7108E38079D027890D13795B6C0825D16DC390092B4EDB52A8E991D46559A1E2973CE7945FB315F3C1EFCBECF44>
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http://purl.uniprot.org/SHA-384/A226B7108E38079D027890D13795B6C0825D16DC390092B4EDB52A8E991D46559A1E2973CE7945FB315F3C1EFCBECF44
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A226B7108E38079D027890D13795B6C0825D16DC390092B4EDB52A8E991D46559A1E2973CE7945FB315F3C1EFCBECF44
http://www.w3.org/2000/01/rdf-schema#comment
"Reductions in GluN2A and GluN2B subunits of NMDA receptors coupled with altered MAPK signaling in response to inflammatory stimuli may be related to the cognitive deficits observed in Alzheimer's disease."
xsd:string
http://purl.uniprot.org/uniprot/#_8D4EA241479386C913C0755D30F124687000B217BC6B10272BEC9144D3F83727DAB4312B941412FFE49A936D23BE22B8
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A226B7108E38079D027890D13795B6C0825D16DC390092B4EDB52A8E991D46559A1E2973CE7945FB315F3C1EFCBECF44
http://purl.uniprot.org/uniprot/Q8CGM3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A226B7108E38079D027890D13795B6C0825D16DC390092B4EDB52A8E991D46559A1E2973CE7945FB315F3C1EFCBECF44
http://purl.uniprot.org/uniprot/#_Q8CGM3-mappedCitation-24761931
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A226B7108E38079D027890D13795B6C0825D16DC390092B4EDB52A8E991D46559A1E2973CE7945FB315F3C1EFCBECF44