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DESCRIBE <http://purl.uniprot.org/SHA-384/A26E1BCAE931466B5CCD4CBB8E6D183D1360BE79A4927D9BF0EC009F19996D652C2A63CC48E1A7582CC462406FEBA3B0>
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http://purl.uniprot.org/SHA-384/A26E1BCAE931466B5CCD4CBB8E6D183D1360BE79A4927D9BF0EC009F19996D652C2A63CC48E1A7582CC462406FEBA3B0
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A26E1BCAE931466B5CCD4CBB8E6D183D1360BE79A4927D9BF0EC009F19996D652C2A63CC48E1A7582CC462406FEBA3B0
http://www.w3.org/2000/01/rdf-schema#comment
"showed that OAC1 treatment led to OCT4-mediated upregulation of HOXB4"
xsd:string
http://purl.uniprot.org/uniprot/#_4D4BCA097106047257B4D1351BE86B796E1B0611ABF035AEA550433CF3341FED64C56337A059708C5B734AC406CAB828
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A26E1BCAE931466B5CCD4CBB8E6D183D1360BE79A4927D9BF0EC009F19996D652C2A63CC48E1A7582CC462406FEBA3B0
http://purl.uniprot.org/uniprot/B5B8Q1
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A26E1BCAE931466B5CCD4CBB8E6D183D1360BE79A4927D9BF0EC009F19996D652C2A63CC48E1A7582CC462406FEBA3B0
http://purl.uniprot.org/uniprot/#_B5B8Q1-mappedCitation-26202933
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A26E1BCAE931466B5CCD4CBB8E6D183D1360BE79A4927D9BF0EC009F19996D652C2A63CC48E1A7582CC462406FEBA3B0