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DESCRIBE <http://purl.uniprot.org/SHA-384/A2D315EAC685AA2C7B25A9EBCE14512B8B41A3B8B006C925E6DE664CC7A3B25423F27C0B11E27324142A11E568015B17>
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http://purl.uniprot.org/SHA-384/A2D315EAC685AA2C7B25A9EBCE14512B8B41A3B8B006C925E6DE664CC7A3B25423F27C0B11E27324142A11E568015B17
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A2D315EAC685AA2C7B25A9EBCE14512B8B41A3B8B006C925E6DE664CC7A3B25423F27C0B11E27324142A11E568015B17
http://www.w3.org/2000/01/rdf-schema#comment
"Results suggest that DNA hypomethylation may be a key mechanism underlying the up-regulation of OCT4 in the recurrence of glioma which facilitates the understanding of the role of stem cells."
xsd:string
http://purl.uniprot.org/uniprot/#_C132875A8E6E26B7A1C54D9268B80BB6E34C918C34202B4D94AA5BCC67B1A6A59ADD35EEB06BCBFE82427F051C27BC15
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A2D315EAC685AA2C7B25A9EBCE14512B8B41A3B8B006C925E6DE664CC7A3B25423F27C0B11E27324142A11E568015B17
http://purl.uniprot.org/uniprot/B5B8P3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A2D315EAC685AA2C7B25A9EBCE14512B8B41A3B8B006C925E6DE664CC7A3B25423F27C0B11E27324142A11E568015B17
http://purl.uniprot.org/uniprot/#_B5B8P3-mappedCitation-26925786
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A2D315EAC685AA2C7B25A9EBCE14512B8B41A3B8B006C925E6DE664CC7A3B25423F27C0B11E27324142A11E568015B17