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DESCRIBE <http://purl.uniprot.org/SHA-384/A3594190B5D88ABC3D71FBDA7D29F7B337A5E9B248E7A8F82451C41E1C0489B2C441F7B5A79BD5DADF8A07D59EAE761A>
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http://purl.uniprot.org/SHA-384/A3594190B5D88ABC3D71FBDA7D29F7B337A5E9B248E7A8F82451C41E1C0489B2C441F7B5A79BD5DADF8A07D59EAE761A
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A3594190B5D88ABC3D71FBDA7D29F7B337A5E9B248E7A8F82451C41E1C0489B2C441F7B5A79BD5DADF8A07D59EAE761A
http://www.w3.org/2000/01/rdf-schema#comment
"These data indicate that LPA increases CCN2 expression through the activation of PKC and PKA. Thus the regulatory functions of the PKC and PKA pathways are implicated in the LPA-induced increase in CCN2 expression"
xsd:string
http://purl.uniprot.org/uniprot/#_4E05D5F1F502574F09848374658687A736433C240802C0120D6D27AF6F7494385478BA38E237360B319445159BB096FA
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A3594190B5D88ABC3D71FBDA7D29F7B337A5E9B248E7A8F82451C41E1C0489B2C441F7B5A79BD5DADF8A07D59EAE761A
http://purl.uniprot.org/uniprot/Q3TQ39
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A3594190B5D88ABC3D71FBDA7D29F7B337A5E9B248E7A8F82451C41E1C0489B2C441F7B5A79BD5DADF8A07D59EAE761A
http://purl.uniprot.org/uniprot/#_Q3TQ39-mappedCitation-26743816
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A3594190B5D88ABC3D71FBDA7D29F7B337A5E9B248E7A8F82451C41E1C0489B2C441F7B5A79BD5DADF8A07D59EAE761A