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DESCRIBE <http://purl.uniprot.org/SHA-384/A427F59C4C3D066750EA9C6D3FC04EB0E7D481072FCE7E63A8BBB8ADBE572EB505944E23CE70EAF3CCE5B9919C796CA1>
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http://purl.uniprot.org/SHA-384/A427F59C4C3D066750EA9C6D3FC04EB0E7D481072FCE7E63A8BBB8ADBE572EB505944E23CE70EAF3CCE5B9919C796CA1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A427F59C4C3D066750EA9C6D3FC04EB0E7D481072FCE7E63A8BBB8ADBE572EB505944E23CE70EAF3CCE5B9919C796CA1
http://www.w3.org/2000/01/rdf-schema#comment
"loss of BBS1 BBS4 or OFD1 led to decreased NF-kappaB activity and concomitant IkappaBbeta accumulation and that these defects were ameliorated with SFN treatment."
xsd:string
http://purl.uniprot.org/uniprot/#_CD9D8C77B0A55718D9EEAABED4AAF4FDF1A45C05CDDB9083BE61F8EE15F109B5066C8145E9E04C7CF61338B9F2A06BD5
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A427F59C4C3D066750EA9C6D3FC04EB0E7D481072FCE7E63A8BBB8ADBE572EB505944E23CE70EAF3CCE5B9919C796CA1
http://purl.uniprot.org/uniprot/A0A0S2Z328
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A427F59C4C3D066750EA9C6D3FC04EB0E7D481072FCE7E63A8BBB8ADBE572EB505944E23CE70EAF3CCE5B9919C796CA1
http://purl.uniprot.org/uniprot/#_A0A0S2Z328-mappedCitation-24691443
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A427F59C4C3D066750EA9C6D3FC04EB0E7D481072FCE7E63A8BBB8ADBE572EB505944E23CE70EAF3CCE5B9919C796CA1