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DESCRIBE <http://purl.uniprot.org/SHA-384/A4519D9517643D92898187D0E33A6D60E6FDDDF4BF2006F4C637A972A84B059BE5EDA1F7B0095DA3EEA0B33A22D8B0C5>
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http://purl.uniprot.org/SHA-384/A4519D9517643D92898187D0E33A6D60E6FDDDF4BF2006F4C637A972A84B059BE5EDA1F7B0095DA3EEA0B33A22D8B0C5
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A4519D9517643D92898187D0E33A6D60E6FDDDF4BF2006F4C637A972A84B059BE5EDA1F7B0095DA3EEA0B33A22D8B0C5
http://www.w3.org/2000/01/rdf-schema#comment
"down-regulation of NKG2D by EBV-induced IDO metabolite provides a potential mechanism by which EBV escapes NKG2D-mediated attack by immune cells."
xsd:string
http://purl.uniprot.org/uniprot/#_3F1B8B1F4C27FAB2164D2200BEFA4C36DCF03CB191F96524BF2C6E8C6ED77AEBC3FFC5AA5C9E360213F6AF27312E7E7C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A4519D9517643D92898187D0E33A6D60E6FDDDF4BF2006F4C637A972A84B059BE5EDA1F7B0095DA3EEA0B33A22D8B0C5
http://purl.uniprot.org/uniprot/P26718
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A4519D9517643D92898187D0E33A6D60E6FDDDF4BF2006F4C637A972A84B059BE5EDA1F7B0095DA3EEA0B33A22D8B0C5
http://purl.uniprot.org/uniprot/#_P26718-mappedCitation-21277902
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A4519D9517643D92898187D0E33A6D60E6FDDDF4BF2006F4C637A972A84B059BE5EDA1F7B0095DA3EEA0B33A22D8B0C5