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DESCRIBE <http://purl.uniprot.org/SHA-384/A798EA26632968A65F06861B2A9E89ED9196BD01C35D9B62BAE663CA7DB86D7FB46222A97FE6B352B5213FCAB64EF0F7>
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http://purl.uniprot.org/SHA-384/A798EA26632968A65F06861B2A9E89ED9196BD01C35D9B62BAE663CA7DB86D7FB46222A97FE6B352B5213FCAB64EF0F7
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A798EA26632968A65F06861B2A9E89ED9196BD01C35D9B62BAE663CA7DB86D7FB46222A97FE6B352B5213FCAB64EF0F7
http://www.w3.org/2000/01/rdf-schema#comment
"MafA-dependent activation of insulin II gene promoters was inhibited in the presence of Smad2/Smad4 or Smad3/Smad4 and a constitutively active TGF-beta type I receptor and this inhibition was dependent upon the presence of MafA."
xsd:string
http://purl.uniprot.org/uniprot/#_EBCBC55988A6676D71CF30A8F680584B0E2318CEC5AC2B182518CA44DCE56AC8C54538C16B263AD7676657D3DFB3DECF
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A798EA26632968A65F06861B2A9E89ED9196BD01C35D9B62BAE663CA7DB86D7FB46222A97FE6B352B5213FCAB64EF0F7
http://purl.uniprot.org/uniprot/Q8CF90
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A798EA26632968A65F06861B2A9E89ED9196BD01C35D9B62BAE663CA7DB86D7FB46222A97FE6B352B5213FCAB64EF0F7
http://purl.uniprot.org/uniprot/#_Q8CF90-mappedCitation-17927952
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A798EA26632968A65F06861B2A9E89ED9196BD01C35D9B62BAE663CA7DB86D7FB46222A97FE6B352B5213FCAB64EF0F7