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DESCRIBE <http://purl.uniprot.org/SHA-384/A89D57F4689D4D1A3C6C7AAB6F59601D13EF8B5F6DF6C5EDB7D46DB9CB233407BFD75604DAC0A0EE1390F6F4BCF5646A>
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http://purl.uniprot.org/SHA-384/A89D57F4689D4D1A3C6C7AAB6F59601D13EF8B5F6DF6C5EDB7D46DB9CB233407BFD75604DAC0A0EE1390F6F4BCF5646A
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A89D57F4689D4D1A3C6C7AAB6F59601D13EF8B5F6DF6C5EDB7D46DB9CB233407BFD75604DAC0A0EE1390F6F4BCF5646A
http://www.w3.org/2000/01/rdf-schema#comment
"The augmentation of FGF2 expression and reduced optokinetic responses during the resolution of surface vasculopathy may indicate a role for FGF2 in the maintenance of neuroretinal function in OIR/ROP."
xsd:string
http://purl.uniprot.org/uniprot/#_8ED85ED7898C0E1492411176B32EE7876038E1AB43EC1EB1AF3E26EA2185F0A2C84829DC1444316947A5D80E6272E90C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A89D57F4689D4D1A3C6C7AAB6F59601D13EF8B5F6DF6C5EDB7D46DB9CB233407BFD75604DAC0A0EE1390F6F4BCF5646A
http://purl.uniprot.org/uniprot/Q925A2
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A89D57F4689D4D1A3C6C7AAB6F59601D13EF8B5F6DF6C5EDB7D46DB9CB233407BFD75604DAC0A0EE1390F6F4BCF5646A
http://purl.uniprot.org/uniprot/#_Q925A2-mappedCitation-25525167
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A89D57F4689D4D1A3C6C7AAB6F59601D13EF8B5F6DF6C5EDB7D46DB9CB233407BFD75604DAC0A0EE1390F6F4BCF5646A