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DESCRIBE <http://purl.uniprot.org/SHA-384/A9E4AC81A2E64B2378CD1B78AA542BF6EE5129F3060026218CF95DD92804E6614175B90A1AE33CC729A7AF9779DBB1A5>
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http://purl.uniprot.org/SHA-384/A9E4AC81A2E64B2378CD1B78AA542BF6EE5129F3060026218CF95DD92804E6614175B90A1AE33CC729A7AF9779DBB1A5
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/A9E4AC81A2E64B2378CD1B78AA542BF6EE5129F3060026218CF95DD92804E6614175B90A1AE33CC729A7AF9779DBB1A5
http://www.w3.org/2000/01/rdf-schema#comment
"Results imply that in acute ischemic conditions CBS is upregulated and activated by truncation causing an increased production of hydrogen sulfide which exacerbate the ischemic injuries"
xsd:string
http://purl.uniprot.org/uniprot/#_BF0432B9FCBCD482071A1F9D63398E2B7F28FB8DE13049E152AA0B6C746927465EF43DB85D2D9A84BB601F6EFC713EED
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/A9E4AC81A2E64B2378CD1B78AA542BF6EE5129F3060026218CF95DD92804E6614175B90A1AE33CC729A7AF9779DBB1A5
http://purl.uniprot.org/uniprot/P32232
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A9E4AC81A2E64B2378CD1B78AA542BF6EE5129F3060026218CF95DD92804E6614175B90A1AE33CC729A7AF9779DBB1A5
http://purl.uniprot.org/uniprot/#_P32232-mappedCitation-25873304
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/A9E4AC81A2E64B2378CD1B78AA542BF6EE5129F3060026218CF95DD92804E6614175B90A1AE33CC729A7AF9779DBB1A5