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DESCRIBE <http://purl.uniprot.org/SHA-384/ABD9AE8F7C3E30F137C5F02F0BD62129C7AE8514D4091723F7593CD57858905B40B822697062C26AA3757623EB29E56F>
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http://purl.uniprot.org/SHA-384/ABD9AE8F7C3E30F137C5F02F0BD62129C7AE8514D4091723F7593CD57858905B40B822697062C26AA3757623EB29E56F
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/ABD9AE8F7C3E30F137C5F02F0BD62129C7AE8514D4091723F7593CD57858905B40B822697062C26AA3757623EB29E56F
http://www.w3.org/2000/01/rdf-schema#comment
"A modest genetic increase in ACE impairs myocardial tolerance to ischemia. ACE level plays a critical role in cardiac ischemia through both kinin and angiotensin mediated mechanisms."
xsd:string
http://purl.uniprot.org/uniprot/#_2A7A2FC0CEEF912C75598D08AD76C498CCABA63CEE01CBE3BDB106FF6D88B65BFBBBBCACE24BDC51452983E06ABDAE36
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/ABD9AE8F7C3E30F137C5F02F0BD62129C7AE8514D4091723F7593CD57858905B40B822697062C26AA3757623EB29E56F
http://purl.uniprot.org/uniprot/Q3TU20
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/ABD9AE8F7C3E30F137C5F02F0BD62129C7AE8514D4091723F7593CD57858905B40B822697062C26AA3757623EB29E56F
http://purl.uniprot.org/uniprot/#_Q3TU20-mappedCitation-20667972
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/ABD9AE8F7C3E30F137C5F02F0BD62129C7AE8514D4091723F7593CD57858905B40B822697062C26AA3757623EB29E56F