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DESCRIBE <http://purl.uniprot.org/SHA-384/ABF3AEEE4A2C193EDB78E5DD963DA434EC289FABE1A5DDB75DD12A1CAF6DAA207375A00B7C866736F048830D17973017>
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http://purl.uniprot.org/SHA-384/ABF3AEEE4A2C193EDB78E5DD963DA434EC289FABE1A5DDB75DD12A1CAF6DAA207375A00B7C866736F048830D17973017
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/ABF3AEEE4A2C193EDB78E5DD963DA434EC289FABE1A5DDB75DD12A1CAF6DAA207375A00B7C866736F048830D17973017
http://www.w3.org/2000/01/rdf-schema#comment
"we found that in HS fibroblasts (HFs) miR-181c and miR-10a were differentially-expressed and targeted uPA and PAI-1 respectively. The production of Type 1 collagen (Col1) was inhibited by miR-181c knockdown or miR-10a overexpression"
xsd:string
http://purl.uniprot.org/uniprot/#_494058E8F5C497620D5BC253F6D59254BCA7D447CE047B36B7BAC2A2191888A8C495079F665D0E1859D25A3A8A57A977
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/ABF3AEEE4A2C193EDB78E5DD963DA434EC289FABE1A5DDB75DD12A1CAF6DAA207375A00B7C866736F048830D17973017
http://purl.uniprot.org/uniprot/Q16053
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/ABF3AEEE4A2C193EDB78E5DD963DA434EC289FABE1A5DDB75DD12A1CAF6DAA207375A00B7C866736F048830D17973017
http://purl.uniprot.org/uniprot/#_Q16053-mappedCitation-25554417
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/ABF3AEEE4A2C193EDB78E5DD963DA434EC289FABE1A5DDB75DD12A1CAF6DAA207375A00B7C866736F048830D17973017