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DESCRIBE <http://purl.uniprot.org/SHA-384/AC1ED1A543F13F36381A1E117ADB9148CC4B87CAB086FD12DA13C0F356CC694B9B68CB6D8CD30A92972429D766AA0E66>
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http://purl.uniprot.org/SHA-384/AC1ED1A543F13F36381A1E117ADB9148CC4B87CAB086FD12DA13C0F356CC694B9B68CB6D8CD30A92972429D766AA0E66
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/AC1ED1A543F13F36381A1E117ADB9148CC4B87CAB086FD12DA13C0F356CC694B9B68CB6D8CD30A92972429D766AA0E66
http://www.w3.org/2000/01/rdf-schema#comment
"LARGE repression is responsible for the defects in dystroglycan-mediated cell adhesion that are observed in epithelium-derived cancer cells and point to a defect of dystroglycan glycosylation as a factor in cancer progression"
xsd:string
http://purl.uniprot.org/uniprot/#_5486B52E06722F6FD3B712719E41C21BD4B827EBA0AA7A92A1369A47E79CABBE71144C21DBC75C59D3CA052D13F8DD17
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/AC1ED1A543F13F36381A1E117ADB9148CC4B87CAB086FD12DA13C0F356CC694B9B68CB6D8CD30A92972429D766AA0E66
http://purl.uniprot.org/uniprot/B7ZB85
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/AC1ED1A543F13F36381A1E117ADB9148CC4B87CAB086FD12DA13C0F356CC694B9B68CB6D8CD30A92972429D766AA0E66
http://purl.uniprot.org/uniprot/#_B7ZB85-mappedCitation-19244252
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/AC1ED1A543F13F36381A1E117ADB9148CC4B87CAB086FD12DA13C0F356CC694B9B68CB6D8CD30A92972429D766AA0E66