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DESCRIBE <http://purl.uniprot.org/SHA-384/AD8F7C5F8099D26AFF2CBA224859F1E4D4CCF7A6A903B72CA66A8B68D249A858B8FD9C6B819A3666ACE161328FBD7B63>
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http://purl.uniprot.org/SHA-384/AD8F7C5F8099D26AFF2CBA224859F1E4D4CCF7A6A903B72CA66A8B68D249A858B8FD9C6B819A3666ACE161328FBD7B63
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/AD8F7C5F8099D26AFF2CBA224859F1E4D4CCF7A6A903B72CA66A8B68D249A858B8FD9C6B819A3666ACE161328FBD7B63
http://www.w3.org/2000/01/rdf-schema#comment
"Results report that oxygen deprivation can activate the autophagic pathway in human cancer cell lines via AMPK activity independent of HIF-1 BNIP3 and BNIP3L."
xsd:string
http://purl.uniprot.org/uniprot/#_0F865B77148FE8AB26F6397FA657F2476BEC17469060394418A085AFC4E734F87273D732B4749CB89C946B3C46B824B0
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/AD8F7C5F8099D26AFF2CBA224859F1E4D4CCF7A6A903B72CA66A8B68D249A858B8FD9C6B819A3666ACE161328FBD7B63
http://purl.uniprot.org/uniprot/O60238
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/AD8F7C5F8099D26AFF2CBA224859F1E4D4CCF7A6A903B72CA66A8B68D249A858B8FD9C6B819A3666ACE161328FBD7B63
http://purl.uniprot.org/uniprot/#_O60238-mappedCitation-18551130
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/AD8F7C5F8099D26AFF2CBA224859F1E4D4CCF7A6A903B72CA66A8B68D249A858B8FD9C6B819A3666ACE161328FBD7B63