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DESCRIBE <http://purl.uniprot.org/SHA-384/AEAE3BFF12DEBB01B4656F11F43FA927879A257ACED861C2AC997BDF7E5C0C07C55E91C1B25E83FDD87CE27F30B58FC3>
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http://purl.uniprot.org/SHA-384/AEAE3BFF12DEBB01B4656F11F43FA927879A257ACED861C2AC997BDF7E5C0C07C55E91C1B25E83FDD87CE27F30B58FC3
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/AEAE3BFF12DEBB01B4656F11F43FA927879A257ACED861C2AC997BDF7E5C0C07C55E91C1B25E83FDD87CE27F30B58FC3
http://www.w3.org/2000/01/rdf-schema#comment
"Together these results suggest that the inhibitory effect of OSM on TGF-beta1-induced CTGF mRNA expression is mainly driven by STAT3 thereby providing a signaling mechanism whereby OSM may contribute to tubulointerstitial protection."
xsd:string
http://purl.uniprot.org/uniprot/#_6A9CC42681D581D4379690C893CF6DB84A74D1A8BCB7F97E27B2B290CFB83B0E88E781661A62E0002367392E7FA14256
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/AEAE3BFF12DEBB01B4656F11F43FA927879A257ACED861C2AC997BDF7E5C0C07C55E91C1B25E83FDD87CE27F30B58FC3
http://purl.uniprot.org/uniprot/Q17RA0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/AEAE3BFF12DEBB01B4656F11F43FA927879A257ACED861C2AC997BDF7E5C0C07C55E91C1B25E83FDD87CE27F30B58FC3
http://purl.uniprot.org/uniprot/#_Q17RA0-mappedCitation-22814105
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/AEAE3BFF12DEBB01B4656F11F43FA927879A257ACED861C2AC997BDF7E5C0C07C55E91C1B25E83FDD87CE27F30B58FC3