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DESCRIBE <http://purl.uniprot.org/SHA-384/AF4DB7EE1F7BCB4EFFBF79BA134785F4F71158A52B6A28F2EDAEA6BFEDA5F82D7695340F9D0996EF602BB09F40864AD4>
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http://purl.uniprot.org/SHA-384/AF4DB7EE1F7BCB4EFFBF79BA134785F4F71158A52B6A28F2EDAEA6BFEDA5F82D7695340F9D0996EF602BB09F40864AD4
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/AF4DB7EE1F7BCB4EFFBF79BA134785F4F71158A52B6A28F2EDAEA6BFEDA5F82D7695340F9D0996EF602BB09F40864AD4
http://www.w3.org/2000/01/rdf-schema#comment
"Both IGF-1Ea and IGF-1Ec are expressed during normal postnatal brain development hypoxia-ischaemia (HI) or hypoxia alone produced increased and prolonged expression of the IGF-1Ec isoform only; IGF-1Ec may play a role in HI pathology"
xsd:string
http://purl.uniprot.org/uniprot/#_87E1C745CBBAD1895F90C54B1CDE409EFF745FCB3902C3B7639D069F7C73C52F0CA3532965EF4C5DF6ADF75B86224810
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/AF4DB7EE1F7BCB4EFFBF79BA134785F4F71158A52B6A28F2EDAEA6BFEDA5F82D7695340F9D0996EF602BB09F40864AD4
http://purl.uniprot.org/uniprot/P08025
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/AF4DB7EE1F7BCB4EFFBF79BA134785F4F71158A52B6A28F2EDAEA6BFEDA5F82D7695340F9D0996EF602BB09F40864AD4
http://purl.uniprot.org/uniprot/#_P08025-mappedCitation-19766709
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/AF4DB7EE1F7BCB4EFFBF79BA134785F4F71158A52B6A28F2EDAEA6BFEDA5F82D7695340F9D0996EF602BB09F40864AD4