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http://purl.uniprot.org/SHA-384/AF87BF04D8BCB19DCF60DC3D5BB3D399B24F7C32B367B2FC347535CFCE5C71C5FE008A60A2381A9EB884F74CE4D19CCBhttp://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/AF87BF04D8BCB19DCF60DC3D5BB3D399B24F7C32B367B2FC347535CFCE5C71C5FE008A60A2381A9EB884F74CE4D19CCBhttp://www.w3.org/2000/01/rdf-schema#comment"The data suggest a common evolutionary origin for both inhibitory mechanisms called decay acceleration and cofactor activity with variable C3b binding through domains at sites ii iii and iv and provide a framework for understanding regulators of complement activation (RCA) disease-related mutations and immune evasion."xsd:string
http://purl.uniprot.org/uniprot/#_ACA3D17836546312ADF5F67340BCB7A396F07B379BA9650C90F9A3B33851D1B85716388BD3BF1B1ED6B7C9957F5247D4http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/AF87BF04D8BCB19DCF60DC3D5BB3D399B24F7C32B367B2FC347535CFCE5C71C5FE008A60A2381A9EB884F74CE4D19CCB
http://purl.uniprot.org/uniprot/V9HWA9http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/AF87BF04D8BCB19DCF60DC3D5BB3D399B24F7C32B367B2FC347535CFCE5C71C5FE008A60A2381A9EB884F74CE4D19CCB
http://purl.uniprot.org/uniprot/#_V9HWA9-mappedCitation-27013439http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/AF87BF04D8BCB19DCF60DC3D5BB3D399B24F7C32B367B2FC347535CFCE5C71C5FE008A60A2381A9EB884F74CE4D19CCB