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DESCRIBE <http://purl.uniprot.org/SHA-384/B02F0078A3B4F38860A09C199123E6829125D09C26B05ED56CD3DF0A6485CA2640BD6BEF34F9409C79E3D79243C7CF53>
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http://purl.uniprot.org/SHA-384/B02F0078A3B4F38860A09C199123E6829125D09C26B05ED56CD3DF0A6485CA2640BD6BEF34F9409C79E3D79243C7CF53
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/B02F0078A3B4F38860A09C199123E6829125D09C26B05ED56CD3DF0A6485CA2640BD6BEF34F9409C79E3D79243C7CF53
http://www.w3.org/2000/01/rdf-schema#comment
"Taken together our data demonstrate the importance of CD40 signaling in the conversion of CTL exhaustion and its ability to enhance PD-1 antagonist action in rescuing exhausted CTLs in chronic infection."
xsd:string
http://purl.uniprot.org/uniprot/#_CB55F483C74CEC5625DE4AB68FC933641928B9EC0FAF268484071C378A466E11C144E0D4E0162A41614AF7EB58D09DFB
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/B02F0078A3B4F38860A09C199123E6829125D09C26B05ED56CD3DF0A6485CA2640BD6BEF34F9409C79E3D79243C7CF53
http://purl.uniprot.org/uniprot/Q544F3
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/B02F0078A3B4F38860A09C199123E6829125D09C26B05ED56CD3DF0A6485CA2640BD6BEF34F9409C79E3D79243C7CF53
http://purl.uniprot.org/uniprot/#_Q544F3-mappedCitation-28153727
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/B02F0078A3B4F38860A09C199123E6829125D09C26B05ED56CD3DF0A6485CA2640BD6BEF34F9409C79E3D79243C7CF53