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DESCRIBE <http://purl.uniprot.org/SHA-384/B0FCE8ECDB8D047AAA6E2F66D24B03AFC5DE06E731AEE9492DAB7B4A3B9F9DB3BC43E92B7AC083B7F66DC200E20AF6BE>
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http://purl.uniprot.org/SHA-384/B0FCE8ECDB8D047AAA6E2F66D24B03AFC5DE06E731AEE9492DAB7B4A3B9F9DB3BC43E92B7AC083B7F66DC200E20AF6BE
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/B0FCE8ECDB8D047AAA6E2F66D24B03AFC5DE06E731AEE9492DAB7B4A3B9F9DB3BC43E92B7AC083B7F66DC200E20AF6BE
http://www.w3.org/2000/01/rdf-schema#comment
"Results suggest a novel role for the DNA glycosylase Neil3 in atherogenesis in balancing lipid metabolism and macrophage function potentially independently of genome-wide canonical base excision repair of oxidative DNA damage."
xsd:string
http://purl.uniprot.org/uniprot/#_61A8FEF2F47283A7857EE6EE4D132E7D4813E263A91E3D1CE4D7A32456BAC9AC56223D9385CFD66FB1C1FD862A7E911A
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/B0FCE8ECDB8D047AAA6E2F66D24B03AFC5DE06E731AEE9492DAB7B4A3B9F9DB3BC43E92B7AC083B7F66DC200E20AF6BE
http://purl.uniprot.org/uniprot/Q8TAT5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/B0FCE8ECDB8D047AAA6E2F66D24B03AFC5DE06E731AEE9492DAB7B4A3B9F9DB3BC43E92B7AC083B7F66DC200E20AF6BE
http://purl.uniprot.org/uniprot/#_Q8TAT5-mappedCitation-27328939
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/B0FCE8ECDB8D047AAA6E2F66D24B03AFC5DE06E731AEE9492DAB7B4A3B9F9DB3BC43E92B7AC083B7F66DC200E20AF6BE